Hepatitis is inflammation of the liver parenchyma, defined histologically by hepatocyte injury and inflammatory infiltrate and clinically by elevated aminotransferases, with or without jaundice. It arises from hepatotropic viruses (hepatitis A, B, C, D and E), other infections, alcohol, drugs and toxins, autoimmune …
Hepatitis is inflammation of the liver parenchyma, defined histologically by hepatocyte injury and inflammatory infiltrate and clinically by elevated aminotransferases, with or without jaundice. It arises from hepatotropic viruses (hepatitis A, B, C, D and E), other infections, alcohol, drugs and toxins, autoimmune processes, and metabolic or steatotic disease. Viral hepatitis is the dominant global cause: the enterically transmitted A and E viruses produce mostly acute, self-limiting illness, whereas the bloodborne B and C viruses can establish chronic infection that progresses to fibrosis, cirrhosis and hepatocellular carcinoma. Diagnosis integrates serology, nucleic-acid testing and confirmatory assays, while management ranges from supportive care to nucleos(t)ide analogues for chronic hepatitis B and direct-acting antivirals achieving sustained virologic response in hepatitis C. Work in this area examines tenofovir treatment of chronic hepatitis B, confirmatory testing of hepatitis C in blood donors, time-limited HCV regimens delivered in prison and outreach settings, the trends and burden of chronic liver disease in hospital cohorts, occupational hepatitis B prevention, and angiogenic markers in hepatocellular carcinoma complicating HCV. The journal publishes peer-reviewed clinical, epidemiological and laboratory research on viral and other forms of hepatitis.
12 peer-reviewed articles, ranked by relevance. Each links to its DOI.
The 12 articles above have been cited 71 times in the scholarly literature. Citation data via OpenAlex and Crossref, updated Jun 2026.
A sample of recent works citing this journal's research on Hepatitis, linking to each citing work.